Thrombosis | wearecellix 2/2

Adenosine Receptor Agonist HE-NECA Enhances Antithrombotic Activities of Cangrelor and Prasugrel

Cellix Technical Team

Jan 23, 20233 min read

Adenosine Receptor Agonist HE-NECA Enhances Antithrombotic Activities of Cangrelor and Prasugrel

Test the effect of HE-NECA and cangrelor on thrombi formation | Platelet Activation and Thrombus Formation In Vitro |

Cardiovascular: Platelets - Thrombosis Adhesion Assay

Cellix Technical Team

Jan 16, 20235 min read

Cardiovascular: Platelets - Thrombosis Adhesion Assay

Novel microflow system | Low flow rates (5 pL min-1 to 10 μL min-1) and shear stress levels

Cellix's VenaFlux Solutions mimic human blood vessels

Cellix Technical Team

Jan 9, 20233 min read

Cellix's VenaFlux Solutions mimic human blood vessels

Quantify cell adhesion, rolling, migration, cell-cell adhesion, platelet aggregation, thrombi ormation - all under shear flow.

Cellix Technical Team

Jul 4, 20223 min read

People are talking about our food

Thrombus formation under flow in vitro | Treatment with cucurbitacin | Pre-incubation of platelets with cucurbitacins

Cellix's ExiGo Microfluidic syringe pump vs. conventional syringe pumps

Cellix Technical Team

Jun 10, 20222 min read

Cellix's ExiGo Microfluidic syringe pump vs. conventional syringe pumps

Selective Adenosine Receptor (AR) ligands modulate AR activity showing promise as clinical candidate

Cellix Technical Team

May 31, 20224 min read

Selective Adenosine Receptor (AR) ligands modulate AR activity showing promise as clinical candidate

Inhibition of human blood platelets aggregation | Inhibition of human blood platelets aggregation | Formation of thrombus in flow conditions

Bleeding diathesis in mice lacking JAK2 in platelets

Cellix Technical Team

May 6, 20223 min read

Bleeding diathesis in mice lacking JAK2 in platelets

The tyrosine kinase JAK2 protein is critical for the JAK/STAT cytokine signaling of hematopoietic cells. The somatic JAK2 V617F mutation...

Inhibition of NADPH oxidase blocks NETosis & reduces thrombosis in heparin-induced thrombocytopenia

Cellix Technical Team

Apr 8, 20224 min read

Inhibition of NADPH oxidase blocks NETosis & reduces thrombosis in heparin-induced thrombocytopenia

Microfluidic studies | Neutrophil-targeted ROS-inhibition assays | Thrombus formation in HIT requires neutrophil-derived ROS

NETosis and thrombosis in vaccine-induced immune thrombotic thrombocytopenia

Cellix Technical Team

Mar 21, 20224 min read

NETosis and thrombosis in vaccine-induced immune thrombotic thrombocytopenia

NETosis markers | Thrombosis induction | VITT antibodies induce thrombosis and thrombocytopenia in vitro

Eucalyptol Inhibits Agonist-induced Platelet Function and Thrombus Formation in Mice

Cellix Technical Team

Feb 25, 20224 min read

Eucalyptol Inhibits Agonist-induced Platelet Function and Thrombus Formation in Mice

Thrombus Formation | Integrin αIIbβ3 Signaling | Granule Secretion in Platelets | Hemostasis

Nanovesicles dissolve blood clots reducing side-effects

Cellix Technical Team

Jan 24, 20224 min read

Nanovesicles dissolve blood clots reducing side-effects

How does the nanovesicle work | Thrombus selectivity under physiological flow conditions | Targeted thrombolysis under flow conditions |

Harnessing seaweed extract to treat ischemic stroke

Cellix Technical Team

Oct 11, 20214 min read

Harnessing seaweed extract to treat ischemic stroke

On this World Thrombosis Day and in advance of World Stroke Day, taking place on 29th October, we’d like to shine a light on a paper recentl

Blood clots in severely ill COVID-19 patients caused by abnormal antibody response

Cellix Technical Team

Aug 23, 20214 min read

Blood clots in severely ill COVID-19 patients caused by abnormal antibody response

New study reveals blood clotting in critically ill COVID-19 may be caused by an excessive immune response rather than the virus itself.

Trapped! Flow studies reveal Thrombosis target

Cellix Technical Team

Oct 13, 20203 min read

Trapped! Flow studies reveal Thrombosis target

The researchers of this study focused on how recruited platelets interact with neutrophils and whether this promotes the onset of Deep Vein